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May 31, 2020
Bria is a Multi-Platform, Multi-Device, Unified Communications and Presence System that offers a full-featured softphone client for iOS, Linux and Windows devices. CounterPath Bria Softphone is a powerful and versatile softphone client for business VoIP.

See also
Comparison of VoIP software
Comparison of VoIP software

References

Category:Telecommunications companies of the United States
Category:VoIP software
Category:Communication software
Category:Software companies of the United StatesMaternal hypothyroidism alters the expression of genes that control maternal and fetal skeletal development in rats.
Maternal hypothyroidism induces major skeletal abnormalities, including vertebral wedging and rib malformations, in fetal rats. We hypothesized that these skeletal malformations are the result of altered expression of maternal and fetal genes that regulate skeletal formation during embryogenesis. To test this hypothesis, maternal hypothyroidism was induced in pregnant Sprague-Dawley rats and fetuses were collected at 18 days post coitus. Hypothyroid rats displayed significant decreases in maternal serum thyroid hormone levels and no detectable placental thyroid hormone transport. Maternal hypothyroidism did not alter placental and fetal growth, or cause alterations in fetal pituitary-thyroid axis expression of thyroid hormone receptor isoforms. However, maternal hypothyroidism significantly increased expression of Twist and Chst5 in maternal long bones and fetal ribs. These genes are critical to control tissue architecture during embryonic development. In contrast to Twist and Chst5, Fgf2 and Fgf7 expression was not altered in maternal long bones or fetal ribs by maternal hypothyroidism. Maternal hypothyroidism was also associated with an increase in expression of Runx2 and suppression of Osterix expression in maternal long bones and fetal ribs. These results provide further evidence that maternal hypothyroidism leads to alterations in maternal and fetal gene expression, which may contribute to the expression of skeletal malformations during embryogenesis.Meta

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